How chronic stress experienced during early development epigenetically programs adult disease risk

Written by MDI Biological Laboratory

Over on Neurology Central, James Coffman will be presenting a webinar on how adult disease risk is linked to psychosocial stress experienced early in life.

Live event: Thursday 13th April 2017
07:00 [PDT] 10:00 [EDT] 15:00 [BST]

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Chonic psychosocial stress contributes significantly to the public health problems endemic to the modern world, many of which have also been linked to chronic inflammation. Epidemiological studies have shown that chronic stress experienced very early in life–even prenatally–increases the risk of developing inflammatory disease in adulthood, including mental health disorders such as anxiety and depression. Chronically elevated glucocorticoid (corticosteroid) signaling is one mechanism hypothesized to account for that correlation. Consistent with this, chronic exposure to elevated corticosteroids during early development has been found to have long term effects on adult behavior and immunoregulation.

Learn more on Neurology Central.